Orgone Energy
Anti Aging Theories/Technology
Scientists identify triggers that lead to Ageing
Apr 22nd
What is interesting here is that the mechanism of Ageing is more like a program triggering the catabolism of the body rather than a general deterioration. It turns out that the protein Sirt6 regulates telomerase production which determines the lenght of the telomeres. The protein also inhibits the production of another protein called NF Beta Kappa B which actively breaks body tissue down. SIRT6 is a cousin of SIRT1 which is also known to have anti-aging effects. SIRT1 is found naturally in Resveratrol in Grapes and Knot weed.
By Richard Alleyne
Researchers have found two connected chemical triggers that regulate the lifespan of cells and believe that, by influencing them, they can increase longevity and reduce the signs of ageing.
The study by Stanford University in California builds on the growing belief that age-related degeneration of tissues, organs and even facial skin, is an active, deliberate process by the body rather than a gradual failure of tired cells.
The team has found two proteins that seem to directly affect ageing. One – known as SIRT6 – which protects against cell ageing and another – known as NF Kappa B – which promotes it.
What is even more exciting is that the study has discovered for the first time that the two substances influence each other – and that manipulating their relative availability in the body could add years to the average life and reduce wrinkles…..
The scientists concluded that SIRT6 and NF Kappa B may work together to help cells age appropriately as the former “puts the brakes” on the latter. “It seems that an important job of SIRT6 is to restrain NF Kappa B and limit the expression of genes associated with ageing,” said Dr Chang.
Cont’ @ Source
Drinking Speeds up Ageing in Cells
Apr 22nd
Again we have evidence that the telomeres determine the age of the body. If systemically, we can reverse telomere shortening, then we have a real visible age reversal effect.
By Richard Alleyne, Science Correspondent
Researchers have found that drinking damages part of the cells that are linked to premature ageing and cancer. They discovered that it causes stress and inflammation to telomeres – the ends of DNA strands that stop them unravelling much like the ends of shoelaces. As people age, telomere length shortens progressively and eventually they are so damaged the cell dies.
The study found alcohol accelerates this process. Since telomere shortening is thought to also increase cancer risk, the researchers speculated that those with shorter telomeres due to heavy alcohol consumption would have an increased risk of cancer.
Cont’ @ Source
RNA Regenerator– increases production of telomerase to reverse telomere deterioration.
Two Proteins Enable Skin Cell Regeneration
Apr 14th
ScienceDaily (Sep. 28, 2009) — Never mind facial masks and exfoliating scrubs, skin takes care of itself. Stem cells located within the skin actively generate differentiating cells that can ultimately form either the body surface or the hairs that emanate from it. In addition, these stem cells are able to replenish themselves, continually rejuvenating skin and hair. Now, researchers at Rockefeller University have identified two proteins that enable these skin stem cells to undertake this continuous process of self-renewal.
RNA Regenerator– Activates an increase in telomerase.
The work, published in Nature Genetics, brings new details to the understanding of how stem cells maintain — and lose — their status as stem cells and are able to specialize into various types of cells. It also further dissects a ubiquitous Rube Goldberg-like pathway whose molecular gears and levers play an important role in activating stem cells to divide and transform into tissue-making cells.
Lead researcher Elaine Fuchs, head of the Laboratory of Mammalian Cell Biology and Development, and first author Hoang Nguyen, a former postdoc in the lab, worked with mice engineered to lack the proteins TCF3 and TCF4, which reside in the nucleus of skin stem cells, where they bind to DNA to turn genes off that would otherwise cause the stem cells to differentiate. They found that without TCF3 and TCF4, all of the layers of the mice’s skin still develop properly, but they cannot be maintained.
Silver generates pluripotent stem cells.
“The epidermal stem cells — one of the types of stem cells in the skin — lose their capacity to self-renew and replace skin cells that have died,” says Nguyen, who is now an assistant
Source
Stem cells can be made from Adult cells
Feb 9th
. Now this article is a bit vague on the method they used to reprogram, adult skin cells and turn them into embryonic stem cells, but the idea implies that any cell can be re-programmed to return to an unprogrammed state. Basically, you are age regressing a cell to return to a high energy state of youth.
UK and Canadian researchers have made an important step towards creating stem cells without having to use embryos.
Astragalus– can activate the growth of stem cells
In a report published this week in Nature magazine, scientists from the Medical Research Council Centre for Regenerative Medicine at the University of Edinburgh and the Mount Sinai Hospital in Toronto, outline how skin cells can be programmed into create stem cells, without the ethical quandary of using embryos.
“This new method of generating stem cells does not require embryos as starting points and could be used to generate cells from many adult tissues, such as a patient’s own skin cells,” said principal author Andras Nagy, senior investigator at Mount Sinai’s Samuel Lunenfeld Research Institute quoted by the Canadian Press.
Though the technology of creating skin cells into stem cells has been accomplished previously, the act of reprogramming the four genes needed for stem cells required viruses, which posed the risk of cancerous damage to the cells.
“These four genes are very potent, very powerful factors, which also if they get loose, they could create problems,” he said. “For example, they are cancerous.”
Study leader Dr Keisuke Kaji, from the Medical Research Council Centre for Regenerative Medicine at the University of Edinburgh, described to the BBC the importance of the breakthrough though admitted it was early days and more needed to be done.
Source
Inflammation and Aging
Jan 17th
When I am sick or have a headache, I feel old and look older. I have seen this time and time again, that there is a drastic and immediate aging effect as a function of acute illness. The mechanism to make the body break down is inflammation. But, what about chronic inflammation, I also notice that when I eat alot of meat , pasta or wheat products that the same effect of aging is taking place. The inflammation is the cause. When I flip back into raw fruits and vegetables diets, I begin to look younger significantly younger. But, what about low-grade chronic inflammation? Our immune system is constantly fighting off latent diseases that are still embedded in our genetics. The effects are chronic sub-clinical inflammation that accelerates or is the main cause of aging.
Simply taking anti-inflammatories are not the answer, since the diseases are still embedded on the genetic code. The play is still going on, even though we are not aware of it. Then use of high energy technologies such as bio-resonance, tachyon and Orgone help assist in the removal of the negative imprints on the genetic code. It is these songs that need to be deleted from the genetic record. Bio-resonance removes negative memory patterns gradually. The loss of negativity triggers a reduction in immune system responses and a repairing of the genetic code. The immune system does respond to negative emotional states in addition to highly acidic foods and toxins. The reduction of inflammatory responses whether due to emotional, mental stresses, viruses, pathogens or food is one of the primary keys to reversing your AGE.
Can We Cure Aging?
Controlling inflammation could be the key to a healthy old age.
by Kathleen McGowan
Hammond is an elite athlete. He works out two hours a day with a trainer, pushing himself through sprints, runs, and strength-building exercises. His resting heart rate is below 50. He’s won three gold medals and one silver in amateur competitions this year alone, running races from 100 to 800 meters. In his division, he’s broken four national racing records. But perhaps the most elite thing about Hammond is his age.
He is 93. And really, there’s nothing much wrong with him, aside from the fact that he doesn’t see very well. He takes no drugs and has no complaints, although his hair long ago turned white and his skin is no longer taut.
His secret? He doesn’t have one. Hammond never took exceptional measures during his long life to preserve his health. He did not exercise regularly until his fifties and didn’t get serious about it until his eighties, when he began training for the Georgia Golden Olympics. “I love nothing better than winning,” he says. “It’s been a wonderful thing for me.” Hammond is aging, certainly, but somehow he isn’t getting old—at least, not in the way we usually think about it.
Health & Medicine / Aging Can We Cure Aging?
Controlling inflammation could be the key to a healthy old age.
by Kathleen McGowan
From the December 2007 issue; published online December 4, 2007
Jim Hammond is an elite athlete. He works out two hours a day with a trainer, pushing himself through sprints, runs, and strength-building exercises. His resting heart rate is below 50. He’s won three gold medals and one silver in amateur competitions this year alone, running races from 100 to 800 meters. In his division, he’s broken four national racing records. But perhaps the most elite thing about Hammond is his age.
He is 93. And really, there’s nothing much wrong with him, aside from the fact that he doesn’t see very well. He takes no drugs and has no complaints, although his hair long ago turned white and his skin is no longer taut.
His secret? He doesn’t have one. Hammond never took exceptional measures during his long life to preserve his health. He did not exercise regularly until his fifties and didn’t get serious about it until his eighties, when he began training for the Georgia Golden Olympics. “I love nothing better than winning,” he says. “It’s been a wonderful thing for me.” Hammond is aging, certainly, but somehow he isn’t getting old—at least, not in the way we usually think about it.
They say aging is one of the only certain things in life. But it turns out they were wrong. In recent years, gerontologists have overturned much of the conventional wisdom about getting old. Aging is not the simple result of the passage of time. According to a provocative new view, it is actually something our own bodies create, a side effect of the essential inflammatory system that protects us against infectious disease. As we fight off invaders, we inflict massive collateral damage on ourselves, poisoning our own organs and breaking down our own tissues. We are our own worst enemy.
This paradox is transforming the way we understand aging. It is also changing our understanding of what diseases are and where they come from. Inflammation seems to underlie not just senescence but all the chronic illnesses that often come along with it: diabetes, atherosclerosis, Alzheimer’s, heart attack. “Inflammatory factors predict virtually all bad outcomes in humans,” says Russell Tracy, a professor of pathology and biochemistry at the University of Vermont College of Medicine, whose pioneering research helped demonstrate the role of inflammation in heart disease. “It predicts having heart attacks, having heart failure, becoming diabetic; predicts becoming fragile in old age; predicts cognitive function decline, even cancer to a certain extent.”
The idea that chronic diseases might be caused by persistent inflammation has been kicking around since the 19th century. Only in the past few years, though, have modern biochemistry and the emerging field of systems biology made it possible to grasp the convoluted chemical interactions involved in bodywide responses like inflammation. Over a lifetime, this essential set of defensive mechanisms runs out of bounds and gradually damages organs throughout the body
Continued At Source
SOD (Superoxide Dismutase) Anti Aging Enzyme
Jan 10th
In the study on Rockfish there are some fish that live over 200 years old. One of the attribute that they have is that there SOD levels do not go down as they get older. The longlived versions of the Rockfish do not show any aging signs, researchers have coined the phrase “Negligible Senescence.”
Superoxide Dismutase (SOD) is an enzyme that repairs cells and reduces the damage done to them by superoxide, the most common free radical in the body. SOD is found in both the dermis and the epidermis, and is key to the production of healthy fibroblasts (skin-building cells).
Source
Studies have shown that SOD acts as both an antioxidant and anti-inflammatory in the body, neutralizing the free radicals that can lead to wrinkles and precancerous cell changes. Researchers are currently studying the potential of superoxide dismutase as an anti-aging treatment, since it is now known that SOD levels drop while free radical levels increase as we age.
Superoxide Dismutase helps the body use zinc, copper, and manganese. There are two types of SOD: copper/zinc (Cu/Zn) SOD and manganese (Mn) SOD. Each type of SOD plays a different role in keeping cells healthy. Cu/Zn SOD protects the cells’ cytoplasm, and Mn SOD protects their mitochondria from free radical damage.
Abnormalities in the copper- and zinc-dependent superoxide dismutase gene may contribute to the development of Amyotrophic Lateral Sclerosis (ALS), or Lou Gehrig’s disease, in some people. ALS is a fatal disease that causes deterioration of motor nerve cells in the brain and spinal cord. It has been theorized that low levels of superoxide dismutase in those with ALS leaves nerve cells unprotected from the free radicals that can kill them, so researchers have been studying the effect of vitamin E and other antioxidant supplements on the progression of this disease. It was hoped that regular doses of antioxidants could make up for the lack of SOD and help neutralize free radicals. Initial studies were promising, and indicated that vitamin E supplementation could potentially slow the progression of ALS, with some researchers claiming that the risk of death from ALS was as much as 62 percent lower in regular vitamin E users compared to nonusers.
Superoxide Dismutase has also been used to treat arthritis, prostate problems, corneal ulcers, burn injuries, inflammatory diseases, inflammatory bowel disease, and long-term damage from exposure to smoke and radiation, and to prevent side effects of cancer drugs. In its topical form, it may help to reduce facial wrinkles, scar tissue, heal wounds and burns, lighten dark or hyperpigmentation, and protect against harmful UV rays.
SOD is found in barley grass, broccoli, Brussels sprouts, cabbage, wheatgrass, and most green plants. The body needs plenty of vitamin C and copper to make this natural antioxidant, so be sure to get enough of these substances in your diet as well. SOD is also available by injection, sublingual oral supplements, enteric-coated pills, and topical creams. However, keep in mind that this substance must be absorbed in the small intestines, so it is important to choose oral supplements that are either enteric coated or sublingual in order to bypass the stomach acid that destroys SOD before it can be absorbed by the body.
Ascensionenergyprogram– Age reversal through the use of quantum-based energy technologies.
Age Reversal Research Humpback Whales Do Not Age
Dec 22nd
Etheric Water– Tachyonized homeopathic water with monoatomic elements
Could Carrots Be The Secret To A Long Life And Sex Appeal?
Dec 14th
ScienceDaily (2007-05-11) — Researchers have found that eating certain plant substances can slow down the rate of aging — and that females prefer mates that will be long-lived. Carotenoids are naturally-occurring yellow and red pigments found in plants. Animals that eat those plants can then use the pigments to make themselves colourful in order to attract mates. But carotenoids are also antioxidants, which improve an animal’s ability to combat oxidative stress and strengthen its immune system.
This latest research has found for the first time that males eating more carotenoids were better able to protect their cells from damage and so lived longer – and that females found these long-lived males particularly attractive. Continued at source.
Nuskin makes a product that increases Caretoid levels called Life-Pak nano.
Centarians have genes that maintain Telomere length
Nov 12th
Another study has shown that Centarians have an active gene that secrete telomerase more efficiently than non-centarians. Of course they are still subjected to the same aging process and shortening of the telomeres. Telomeres provide information for the DNA to split. As the lenght of Telomeres shortens the rate at which DNA splits slows down and errors crop up more frequently in the code. With saturation of telomerase is should be possible for a complete regeneration of the Genetic code. Researchers are afraid of the implications of their own work. Life extension of several hundred years in the prime of Youth. Will the best discoveries be kept for the elite? RNA regenerator is a supplement design to produce telomerase in the body.
AscensionEnergy– The use of Quantum energy for re-booting the genetic code.
LiveScience.com
Scientists have zeroed in on one apparent key to long life: an inherited cellular repair mechanism that thwarts aging and perhaps helps prevent disease. Researches say the finding could lead to anti-aging drugs.
The study involves telomeres, the ends of chromosomes that have been likened to the plastic tips that prevent shoelaces from unraveling. Telomeres were already known to play a key role in aging, and their discovery led to this year’s Nobel Prize in medicine.
The new study, which focused on Ashkenazi Jews, finds those who lived the longest had inherited a hyperactive version of an enzyme called telomerase that rebuilds telomeres.
In effect, centenarians tend to have a top-notch body mechanic at work 24/7 repairing the hardware that runs the body, versus a normal person whose body’s cellular control center is left to wear out with time.
“Humans of exceptional longevity are better able to maintain the length of their telomeres,” said Yousin Suh, associate professor of medicine and of genetics at the Albert Einstein College of Medicine at Yeshiva University. “And we found that they owe their longevity, at least in part, to advantageous variants of genes involved in telomere maintenance.”
The results are detailed this week in the Proceedings of the National Academy of Sciences.
Telomeres are short bits of specialized DNA that cap the chromosomes, which tell a cell what to do. Over time, cells divide over and over to keep the body alive. But with each cell division, telomeres get shorter. When they become too short, the cell stops dividing and lapses into a state called cell senescence. Vital tissues are no longer produced, and organs start to fail.
All this was known, and telomeres have been a focus of anti-aging research for years. However, no silver bullets have been discovered to increase the average lifespan.
In the new study, Suh and colleagues studied Ashkenazi Jews, a homogeneous population whose genetics are well-studied. Three groups were part of the research: A very old (average age 97) but healthy group of 86 people; 175 of their offspring; and a control group of 93 offspring of parents who lived a normal lifespan.
“Our research was meant to answer two questions,” explained said Einstein researcher Gil Atzmon in a statement. “Do people who live long lives tend to have long telomeres? And if so, could variations in their genes that code for telomerase account for their long telomeres?”
“Yes” on both accounts, the scientists conclude.
The old crowd had “inherited mutant genes that make their telomerase-making system extra active and able to maintain telomere length more effectively,” the researchers write. “For the most part, these people were spared age-related diseases such as cardiovascular disease and diabetes, which cause most deaths among elderly people.”
“Our findings suggest that telomere length and variants of telomerase genes combine to help people live very long lives, perhaps by protecting them from the diseases of old age,” Suh said. “We’re now trying to understand the mechanism by which these genetic variants of telomerase maintain telomere length in centenarians. Ultimately, it may be possible to develop drugs that mimic the telomerase that our centenarians have been blessed with.”
Thanks to Chris finding this info. Emergency retirement plans
Pig Bladder Powder Regenerates Human Finger
Oct 19th
Posted by Mark Frauenfelder, March 24
A man cut off his finger tip while working on a model plane. His brother, a medical research scientist, sent him a vial containing powdered pig bladder and told him to sprinkle on the severed finger tip. It grew back — “flesh, blood, vessels and nail” — in four weeks.
That powder is a substance made from pig bladders called extracellular matrix. It is a mix of protein and connective tissue surgeons often use to repair tendons and it holds some of the secrets behind the emerging new science of regenerative medicine.
“It tells the body, start that process of tissue regrowth,” said Badylak.
Badlayk is one of the many scientists who now believe every tissue in the body has cells which are capable of regeneration. All scientists have to do is find enough of those cells and “direct” them to grow.
“Somehow the matrix summons the cells and tell them what to do,” Badylak explained. “It helps instruct them in terms of where they need to go, how they need to differentiate – should I become a blood vessel, a nerve, a muscle cell or whatever.”
Source
Orgone Zapper– Simple and inexpensive answers to health, boost immune system kill parasites and viruses.